Điều trị thành công bệnh nhân bỏng acid chromic có biến chứng tổn thương thận cấp

Abstract

Chemical burns appeared when people knew how to use chemicals and began to thrive in the 15th - 16th centuries. With the development of science and technology and industry, chemical burns become more common. The rate of chemical burns is third only to burns caused by dry heat and wet heat [1]. Circumstances causing chemical burns are very diverse: due to domestic accidents; in people exposed to chemicals such as in laboratories, chemical industries, construction and non-professional use of chemicals; involved in criminal acts and suicide; in which, chemical burns related to labor account for a high proportion (1/2 - 2/3 of the total number of chemical burn patients) [1], [2].
Chemical burns include acid burns and base burns. Mechanistically, base burns often cause liquefaction necrosis and saponification, causing chemicals to continue to penetrate deeply, causing aggravation of injury, while acid burns tend to produce dry and coagulated necrosis. Prevents deep penetration of chemicals into subcutaneous tissues [2], [3]. However, there are still some acids that can penetrate deeply to cause toxicity to the body such as formic, chromic, muriatic, sulfuric acids, etc. Vulnerable organs are usually organs with elimination functions such as the liver and kidney [1].
In this report, we would like to introduce a clinical case of chromic acid burn patient with complications of acute kidney injury who was successfully treated.